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What actually causes Pimples you must know

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What actually causes Pimples you must know

Acne is a persistent sebaceous gland condition. While it may appear that pimples form suddenly, the development of an acne outbreak is a lengthy process that begins at the cellular level.

All pimples start with a blocked hair follicle or pore. Understanding why a blockage forms and how a pimple forms can eventually aid you in curing your acne.

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How is the skin layers affect the breakout of pimples?

The epidermis


The epidermis is the skin’s outermost layer, the portion you see every day. The epidermis is made up of five layers.

The stratum germinativum is the epidermis’s lowest layer. It is made up of only one layer of cells. Mitosis, or cell division, occurs in the stratum germinativum.

The new cells begin their journey to the skin’s surface by ascending through the epidermis. They are pushed up through the stratum mucosal first, followed by the stratum spinosum. The base layer is made up of these three layers (stratum germinativum, mucosal, and spinosum).

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The stratum granulosum is reached as the skin cells migrate farther. The cells in this layer begin to die and harden, giving them a granular look. The cells are dead by the time they reach the stratum corneum.

The stratum corneum is made up of these densely packed dead skin cells, which are constantly shed and replaced.

This is known as desquamation. From cell birth to sloughing off, the process takes around 28 days.

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The dermis

The dermis is the deepest, or innermost, layer of your skin, and is composed of tight connective tissue. The dermis feeds and circulates blood to the epidermis. It is also responsible for the skin’s suppleness.

The dermis is substantially thicker than the epidermis, yet it only contains two distinct layers: papillary and reticular.


Blood and lymph arteries, nerves, arrector pili muscles (the muscles that cause your hair to stand on end), sudoriferous (sweat) glands, sebaceous (oil) glands, and hair follicles are all found inside the dermis.

Acne starts in the hair follicles and sebaceous glands.

The Follicle of Hair


Hair follicles are little tube-like openings in the skin that allow hair and sebum to reach the skin’s surface.

The pore entrance, hair root and bulb, sebaceous duct, and sebaceous gland are all components of the follicle.
The epidermis lines the hair follicle’s interior while enclosed within the dermis.

The sebaceous glands release oil, or sebum, into the pore of a normally functioning follicle. Sebum and dead cells shed from the stratum corneum often appear at the skin’s surface via the pore opening.

This approach, however, fails in acne patients.

Sebum and dead skin cells are readily caught within the follicles of acne-prone skin. This buildup of cellular detritus and sebaceous matter hardens and obstructs the pore opening.

A comedo is a type of barrier. It appears on the skin’s surface as a non-inflamed lump or blackhead.

Acnes Propionibacteria


The bacteria Propionibacterium acnes (P. acnes) is responsible for inflammatory acne outbreaks.

P. acnes is a common inhabitant of the hair follicle. They are usually harmless. P. acnes, on the other hand, proliferate uncontrollably in acne-prone skin.

When a comedone obstructs the pore entrance, an anaerobic condition, or a shortage of oxygen, is created within the follicle.

This anaerobic environment, along with abundant sebum within the pore, promotes the growth of P. acnes bacteria.

The follicle swells as it fills up with sebum, dead cells, and germs. The follicular wall ruptures, and the contents leak into the dermis.

To combat the infection, white blood cells rush in. There is swelling and redness, as well as the formation of pus. A pimple has appeared.

If the follicular wall rupture occurs near the surface, the pimple is generally small and heals fast.

More serious lesions, such as nodules and cysts, develop when the rupture occurs deep into the dermis.

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